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* In patients who develop secondary pneumonias of a bronchopneumonia pattern---many bacteria,such as staphylococcus aureus, gram-negative bacilli * Lobular pneumonia is characterized by foci of acute suppurative inflammation centered on bronchioles. The consolidation may be patchy through one lobe but is more frequently multilobar and frequently bilateral and basal The lesions are gray-red to yellow, and up to 0.5 to 1 cm in diameter. Confluence of these foci producing confluent bronchopneumonia. Dry, granular, gray-red to yellow, and poorly delimited at the their margins They vary in size up to 0.5 to 1 cm in diameter. Confluences of these foci occurs in the more florid instances * Suppurative exudate fills the brochi and adjacent alveolar spaces Neutrophils are dominat in this exdation. * * Pleura involvement: the pleura is involved, the inflammation spread to the pleura Bacteremia: is the most serious complication of pneumococcal penumonia.---death, meningitis and endocarditis Suppuration: liquefactive necrosis of alveoli leading to areas of destryoed lung replaced by pus Necrotizing bacterial pneumonia: rare complication, extremly severe necrosis of the lung associated with rapidly progressive disease with a high mortality rate. Pulmonary carnification: the exudation is taken place by granulation tissue It may convert part of lungs into solid tissue. Acute interstitial pneumonia results from infection by agents that are predominantly obligate intracellular pathogens. Infection with these pathogens evokes an acute inflammation that is usually restricted to the interstitum wihtout involvement of the alveolar spaces. * The septa is expanded by hyperemia, edema, and a cellular infiltrate composed of lymphocytes and plasma cells Inclusion bodies: cytomegalovirus * * Hayline membrane Expanded septum Necrosis of epithelial Inclusion bodies * Cornonavirus, a new member of the family coronviridae * In the end of 2002 to 2003, SARS came to be epidemic all over the world
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