天津医科大学内科学课件 Individual Cardiac Arrhythmias.pptVIP

The important content(HBP) 1.CLASSIFICATION OF BLOOD PRESSURE FOR ADULTS AGED 18 YEARS AND OLDER (Table-1) 2.Renin-Angiotensin System(RAS) 3.Target Organ Damage : (1) FUNDUSCOPIC EXAMINATION (2) CARDIAC INVOLVEMENT (3) RENAL FUNCTION (4) CEREBRAL INVOLVEMENT 4.ANTIHYPERTENSIVE DRUG THERAPY (1) the mechanisms of five classes of anti-hypertensive drugs (2) the main side effects 5.The goal of antihypertensive therapy FIGURE 25–4. A, Sinus tachycardia (150 beats/min) in a patient during acute myocardial ischemia; note the ST segment depression. P waves are indicated by arrows. B, Sinus bradycardia at a rate of 40 to 48 beats/min. The second and third QRS complexes (arrows) represent junctional escape beats. Note the P waves at the onset of the QRS complex. C, Nonrespiratory sinus arrhythmia occurring as a consequence of digitalis toxicity. Monitor leads. FIGURE 25–4. A, Sinus tachycardia (150 beats/min) in a patient during acute myocardial ischemia; note the ST segment depression. P waves are indicated by arrows. B, Sinus bradycardia at a rate of 40 to 48 beats/min. The second and third QRS complexes (arrows) represent junctional escape beats. Note the P waves at the onset of the QRS complex. C, Nonrespiratory sinus arrhythmia occurring as a consequence of digitalis toxicity. Monitor leads. CD Figure 240. Atrial fibrillation. A, Untreated. Note the regular fibrillatory waves (best seen in V1) and the rapid ventricular response at rates up to 170 beats/minute. QRS morphology is normal, confirming that this is an atrial arrhythmia. B, Treated with digoxin. Here, the ventricular rate is slower because of digoxin therapy. Note the widespread ST and T wave changes with characteristic reversed-tick configuration. These changes are caused by digoxin and do not necessarily indicate toxicity. They make interpretation of ST change during stress testing almost impossible. C, Paroxysmal atrial fibrillation. After the first two sinus beats, there is a paro