植物激素信号通路.docxVIP

一 脱落酸The simple model of ABA signal in stomatal guard cells. Net effect is the lose of potassium ion and the anion （Cl -so on）in cells. cADPR----Cyclic ADP Ribose. IP3---- inositol(1,4,5)-trisphosphat.NO---- nitric oxide. PA---- Acide Phosphatidique. PLC---- Phospholipase C. PLD---- Phospholipase D. R---- receptor. ROS---- reactive oxygen species. S1P---- Sphingosine 1-phosphate. ①Biding of ABA and receptor(in order to see better, only show the extracellular receptor); ②After ABA combining receptor, the ROS is induced to produce. Then the Ca+ channels is activated on the plasma membrane, ROS is produced by PID which mediated phosphatidic acid; ③The Ca+ internal flow caused instantaneous changes of intracellular calcium. That promoted the release of calcium from vacuoles. ④ ABA is to stimulate the product of NO, and NO add cADPR. ⑤ABA add IP3 by including S1p, the heterogeneous co-polymer and PLD,PLC. ⑥The rise of cADPR and IP3 activate other calcium channel of the vacuole membrane, more CA+ release from vacuoles.⑦The rise of intracellular calcium blocking the channels of K+ on the plasma membrane.⑧The rise of intracellular calcium to promote open Cl- channels on the plasma membrane⑨Proton pump on the plasma membrane is restrained by ABA induced of intracellular calcium increase and inhibition of intracellular pH increased, plasma membrane depolarization further.⑩K+ and anion from across the plasma membrane vacuole first released into the cytoplasm.二 生长素Auxin and TIR1/ABF receptor activation after auxin response model of gene transcription。1. In the absence of auxin, AUX /IAA deter that the ARF transcription activation combined with the formation of inactive form to suppress the auxin induction of gene transcription. Auxin and SCFTIR1 united that promote them interaction with AUX/IAA.2. Auxin activated SCFTIR1 complex whole made the ubiquitin molecules linked to AUX/IAA. Promoting AUX/IAA degrade by protease. AUX/IAA maked the ARF transcriptional activation protein