Recent prevention and treatment of vascular dementia.docVIP

Recent prevention and treatment of vascular dementia.doc

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Recent prevention and treatment of vascular dementia

 PAGE \* MERGEFORMAT 22 Recent prevention and treatment of vascular dementia [Abstract] With the growing elderly population, China has gradually entered into the age-related social and clinical vascular dementia is also increasing, vascular dementia etiology and pathogenesis of complex treatment also varied, now on its prevention and Recent treatment are reviewed. [Keywords:] the pathogenesis of vascular dementia prevention of drug treatment of non-drug treatment [Abstract] With the growing elderly population, China has gradually entered the age-social, clinical Shang vascular dementia is also increasing, vascular dementia etiology, mechanism Fuza, Zhiliao instruments also varied, taking the current situation is reviewed their treatment. [Keywords:] Vascular dementia; Pathogenesis; Prevent, Drug therapy, Non-drug treatment Vascular dementia (Vascular Dementia, VD is due to acute or chronic cerebral vascular disease due to persistent brain dysfunction resulting from comprehensive cognitive impairment, based on language, memory, visual spatial skills, emotion, personality, and computing power, the abstract sense of cognitive impairment such as dementia syndrome. research shows that, VD incidence increases with age, the incidence of VD in China accounts for about 60% of all dementia patients, is China a major component of senile dementia section. Because the pathogenesis of VD is more complex, in the event, there is no particularly effective treatment for VD treatment at home and abroad were a lot of research and exploration, we now present a variety of prevention and treatment methods are summarized as follows: Etiology and pathogenesis 1 VD cause a variety of types, the most common is cerebral infarction. Infarction of the brain involved in cognitive function key parts of the neuron necrosis, apoptosis, and absence of subcortical cerebral ischemia, hypoxia, metabolic changes in brain function and hypoperfusion of the central cortex,

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