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03 gum disease(国外英文资料)
03 gum disease(国外英文资料)
Gum disease. TXT gets up every morning to see the Forbes rich list, and if it doesnt have my name, I go to work. Talking about money doesnt hurt feelings. I curse your buying instant noodles without getting flavoring bags. Gingival disease Gingival Diseases
Chronic gingivitis
An increase in gingivitis
A pregnancy gingivitis
The pregnancy of the gums
The gingivitis
Acute gum papillitis
Drug gum hyperplasia
The disease of gum fibrosis
The gum disease of leukemia is damaged
Acute multiple gums abscess
Low gum tumour
A necrotic ulcerative gum disease
Vitamin C deficiency of gingivitis
Chronic gingivitis
Gingivitis Gingivitis is only associated with dental plaque
Borderline Gingivitis
Simple Gingivitis Simple Gingivitis
A. no other local stimulant
B. accompanied by local stimulant
Prevalence is widespread
The prevalence of children and adolescents is 70-90%
Puberty peaks
The cause of
Plaque - initiating factor
Dental rock, food embedded, bad repair body - local promotion factors
Clinical pathology
Initial lesion was initial lesion
Time: 2-4 days
Clinical: can be healthy gums
Pathology: vasodilation, gingival sulcus fluid (GCF), white blood cells (mainly for the PMNs) bleeding from the wall to the combination of epithelium and gingival sulcus zone, inflammatory infiltration area accounts for about 5% of the connective tissue
Early lesions early lesion
Time: 4-7 days
Clinical: the gums are red and swollen
Pathology: angiogenesis expands. Mainly lymphocyte (about 75%), and neutrophils infiltrate, inflammation is 15%, collagen is destroyed, white blood cells devour bacteria, epithelium is stapled.
Establish an established lesion
Time: chronic gum inflammation
Clinical: the gums are clearly inflamed, red and swollen, and the bleeding of the gingiva deepens
Pathology: plasma cells (Page Schroeder), venous return, gum hypoxia, inflammation area loss of collagen, leukocyte infiltration, epithelial spikes significantly, but the epithelial attachment positi
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