病理问答(国外英文资料).docVIP

病理问答(国外英文资料) 1, the basic pathological changes of atrophy. Answer: (1) the reduction of volume or substance (2) the change of shape can not be obvious (3) the weight loss (4) the color becomes deeper (5) the texture becomes hard. Photoscope: (1) the volume of the actual cell shrinks, and the quantity decreases (2) the hyperplasia. Electron microscopy (1) the degeneration of the organelles, reducing (2) the small amount of autophagy (3) lipids. 2, the main causes of cell edema, pathogenesis, good organ and pathological characteristics. Answer: reason: lack of oxygen, infection, poisoning. Mechanism: sodium potassium pump dysfunction. Good hair organ: liver heart kidney. Pathological features: increased volume, light color; Lens: the cell is swollen, the cytoplasm is light, bright, the nucleus is slightly larger; Electron microscopy: the swelling of the mitochondria, endoplasmic reticulum. 3, the causes of liver fat degeneration, pathogenesis, pathological changes and consequences. Answer: reason: lack of oxygen, infection, poisoning, nutrient deficiency; Mechanism: (1) the increase of fatty acid in liver cells; (2) the synthesis of triglycerides; (3) the apolipoprotein decreased. Pathological changes: the human eye: the liver is big, the edges blunt, the color is light yellow, the quality is soft, the oily feeling; Photoscope: the stem cell is large, the cytoplasm has a round or ovoid cavity, which can squeeze the nucleus on one side, the liver is disturbed, the livers sinus is distorted, the stenosis is obliterated; Consequence: heavy liver cells necrosis, secondary fibrosis. 4, for example, how can the transparent degeneration of the connective tissue of fibers occur, and what is its morphological performance? A: what is happening: the physiologic and pathologic hyperplasia of fibrous connective tissue, such as atrophy of ovarian, breast, chronic sclerosing glomerulonephritis, renal infarction after machine etc. Morphology: white, white, translucent, and tough; Le