Interleukin-13 Upregulates Eotaxin Expression in Airway Epithelial Cells by a STAT6-Dependent Mechanism:(Interleukin-13上调Eotaxin STAT6-Dependent表达式在气道上皮细胞的机制).pdfVIP

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Interleukin-13 Upregulates Eotaxin Expression in Airway Epithelial Cells by a STAT6-Dependent Mechanism:(Interleukin-13上调Eotaxin STAT6-Dependent表达式在气道上皮细胞的机制).pdf

Interleukin-13 Upregulates Eotaxin Expression in Airway Epithelial Cells by a STAT6-Dependent Mechanism:(Interleukin-13上调Eotaxin STAT6-Dependent表达式在气道上皮细胞的机制)

Interleukin-13 Upregulates Eotaxin Expression in Airway Epithelial Cells by a STAT6-Dependent Mechanism Satoshi Matsukura,* Cristiana Stellato, Steve N. Georas, Vincenzo Casolaro, James R. Plitt, Katsushi Miura, Shin Kurosawa, Ulrike Schindler, and Robert P. Schleimer Johns Hopkins Asthma and Allergy Center, Baltimore, Maryland; and Tularik, Inc., South San Francisco, California Interleukin (IL)-13 is a T helper 2–derived cytokine that has re- phils and mononuclear cells, epithelial damage, hyperpla- cently been implicated in allergic airway respo

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