genetic mismatches between nuclei and mitochondria make yeast hybrids sterile细胞核与线粒体基因不匹配使酵母混合动力车无菌.pdfVIP
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genetic mismatches between nuclei and mitochondria make yeast hybrids sterile细胞核与线粒体基因不匹配使酵母混合动力车无菌
Synopsis
Genetic Mismatches Between Nuclei and Mitochondria
Make Yeast Hybrids Sterile
Robin Meadows*
Freelance Science Writer, Fairfield, California, United States of America
When one species mates with another,
the resulting hybrids typically die or fail to
reproduce. Hybrids can form between
similar species from microorganisms to
mammals, with the mule being a classic
example. Sterile offspring of horses and
donkeys, mules are prized for being more
agile than the former, less obstinate than
the latter, and smarter than both. But
researchers prize hybrids for an additional
reason: these reproductive dead ends
could help explain how new species begin
to form. A critical mechanism on the path
to speciation is known as genetic incom-
patibility, in which genes from diverging
species no longer interact properly. These
improper interactions prevent these spe-
cies from producing fertile offspring,
leading to reproductive isolation.
Understanding the basis of genetic
incompatibility is key to identifying the
driving forces of speciation. In a new study
in this issue of PLoS Biology, Jun-Yi Leu
and colleagues address the role of incom-
patibility between genomes in the nucleus Is the fast-evolving mitochondrial genome a driving force of speciation? Image: S-L
and mitochondria in generating reproduc- Chang.
doi:10.1371/journal.pbio.1000433.g001
tive isolation in yeast.
Mitochondria have a limited genome
that produces a mere eight of the 1,000-
In this study, Leu and colleagues set out ing cytonuclear incompatibility. The worst
some proteins this organelle needs to
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