促红细胞生成素对窒息新生大鼠脑促凋亡蛋白Omi%2fHtrA2表达地影响.pdf

in which rat control isstill the96rats group healthy,while hypoxieinjury,45 overand52ratsBecomecouldnotscreamwhentheir couldnotturnthemselves tailsare are32ratscouldn’tturnthemselvesandbecomecould gripped．There notscreamwhentheirtailsare ofbrain griPPed．(2)morphologicalchanges by had HE Neuronsofcontrol ratswere a staining group elativelylarge，each locatednucleussurrounded centrally by large，round with nucleusdistinctnucleolusandnuclear was cytoplasm．The envelop stained．Nissl’Sinthesecells as thin pale—blue body appearedvery cellscouldbeseenwiththeir granules．Some processes．：asphyxiagroup： neuronsbecamesmallerandshrinkedinto cells，some infiltratinginflammatory or andnucleiweredark—blue polygonalveryirregularshape，whosecytoplasm in stained，obscurestructure；(3)Omi／HtrA2 protein cellsshowed and locatedinnervecell staining positive brown-yellowmainly and controlhavesmallnumberofOmi／HtrA2 processes，thegroup cytoplasm ofOmi／HtrA2 in proteinexpression，Theexpression proteinasphyxiagroup increasedat6handreachedthe