distinct rgk gtpases differentially use α1- and auxiliary β-binding-dependent mechanisms to inhibit cav1.2cav2.2 channels不同rgk gtpase不同使用α1u2014u2014和辅助β-binding-dependent机制来抑制cav1.2cav2.2频道.pdfVIP

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distinct rgk gtpases differentially use α1- and auxiliary β-binding-dependent mechanisms to inhibit cav1.2cav2.2 channels不同rgk gtpase不同使用α1u2014u2014和辅助β-binding-dependent机制来抑制cav1.2cav2.2频道.pdf

distinct rgk gtpases differentially use α1- and auxiliary β-binding-dependent mechanisms to inhibit cav1.2cav2.2 channels不同rgk gtpase不同使用α1u2014u2014和辅助β-binding-dependent机制来抑制cav1.2cav2.2频道

Distinct RGK GTPases Differentially Use a - and Auxiliary 1 b-Binding-Dependent Mechanisms to Inhibit CaV 1.2/ CaV2.2 Channels Tingting Yang*, Akil Puckerin, Henry M. Colecraft* Department of Physiology and Cellular Biophysics, Columbia University, College of Physicians and Surgeons, New York, New York, United States of America Abstract Ca 1/Ca 2 channels, comprised of pore-forming a and auxiliary (b,a d) subunits, control diverse biological resp

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distinct rgk gtpases differentially use α1- and auxiliary β-binding-dependent mechanisms to inhibit cav1.2cav2.2 channels不同rgk gtpase不同使用α1u2014u2014和辅助β-binding-dependent机制来抑制cav1.2cav2.2频道.pdfVIP