植物激素——水杨酸.ppt

The panel on the top left seems a bit counter-intuitive, but in this case the HDA19 is silencing an inhibitor of PR-1, so overexpression of HDA19 cause a more vigorous defense response and more resistance. * These two papers came out back-to-back and really made the case for SA as an endogenous signal in defense. * Thanks to the NSF for this beautiful illustration! * On the left h.p.i. is hours post infection. On the right, exudate from dir-1 mutants fails to induce PR-1 gene expression in wild-type plants, but the mutants respond to wild-type exudate. * http://www.ifr.ac.uk/protall/NewsletterAug2000.html * There are some fun videos of arum lilies on the Teaching Tools YouTube channel (/user/PlantTeachingTools) * Grafting studies support MeSA as a systemic signal SA CH3 MeSA SAMT SABP2 SA methyl transferase (SAMT) methylates SA SA binding protein 2 (SABP2) demethylates MeSA Grafts were made between SAMT-silenced plants and wild type SAMT-silenced plants cannot make MeSA From Park, S.-W., Kaimoyo, E., Kumar, D., Mosher, S., and Klessig, D.F. (2007). Methyl salicylate is a critical mobile signal for plant systemic acquired resistance. Science 318: 113-116. Reprinted with permission from AAAS. Grafting studies support MeSA as a systemic signal From Park, S.-W., Kaimoyo, E., Kumar, D., Mosher, S., and Klessig, D.F. (2007). Methyl salicylate is a critical mobile signal for plant systemic acquired resistance. Science 318: 113-116. Reprinted with permission from AAAS. SAMT (hence MeSA) is necessary for SAR Control grafts Experimental grafts SAMT (hence MeSA) is necessary in infected leaves but not systemic leaves W is wild-type S is SAMT-silenced SAMT SA MeSA SAR requires SABP2 in systemic but not infected leaves Control grafts Experimental grafts SAR requires conversion of MeSA to SA at the systemic site MeSA SABP2 SA SABP2-silenced plants cannot convert MeSA back to SA W is wild-type S is SABP2-silenced From Park, S.-W., Kaimoyo, E., Kumar, D., Mosher, S., and