Serum resistin levels in critically ill patients are associated with inflammation, organ dysfunction and metabolism and may predict survival of non-septic patients 英文参考文献.docVIP

Serum resistin levels in critically ill patients are associated with inflammation, organ dysfunction and metabolism and may predict survival of non-septic patients 英文参考文献.doc

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Serum resistin levels in critically ill patients are associated with inflammation, organ dysfunction and metabolism and may predict survival of non-septic patients 英文参考文献

Available online /content/13/3/R95 Research Open Access Vol 13 No 3 Serum resistin levels in critically ill patients are associated with inflammation, organ dysfunction and metabolism and may predict survival of non-septic patients Alexander Koch1*, Olav A Gressner2*, Edouard Sanson1, Frank Tacke1* and Christian Trautwein1* 1Department of Medicine III, RWTH-University Hospital Aachen, Pauwelsstrasse 30, 52074 Aachen, Germany 2Institute of Clinical Chemistry and Pathobiochemistry, RWTH-University Hospital Aachen, Pauwelsstrasse 30, 52074 Aachen, Germany * Contributed equally Corresponding author: Alexander Koch, akoch@ukaachen.de Received: 2 Apr 2009 Revisions requested: 14 May 2009 Revisions received: 27 May 2009 Accepted: 19 Jun 2009 Published: 19 Jun 2009 Critical Care 2009, 13:R95 (doi:10.1186/cc7925) This article is online at: /content/13/3/R95 ? 2009 Koch et al.; licensee BioMed Central Ltd. This is an open access article distributed under the terms of the Creative Commons Attribution License (/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Abstract Introduction Blood glucose levels and insulin resistance in critically ill patients on admission to intensive care units (ICUs) have been identified as factors influencing mortality. The pathogenesis of insulin resistance (IR) in critically ill patients is complex and not fully understood. Resistin is a hormone mainly derived from macrophages in humans and from adipose tissue in rodents, which regulates glucose metabolism and insulin sensitivity. In non-critically ill patients, resistin was found to be related to impaired glucose tolerance, insulin resistance, metabolic syndrome, obesity and type 2 diabetes. Therefore, resistin might represent a link between inflammation, acute phase response and insulin resistance in critically ill patients. We aimed t

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