呼酸基本常识(国外英文资料).docVIP

呼酸基本常识 The pH may stay in the normal range. Therefore, the chronic respiratory acidosis of 10, 6 kPa in PaCO2 is often a compensatory acidosis, and the clinic does not correct it. If PaCO2 is more than 10.6 kPa, the plasma PH will not return to normal even if the kidney has the maximum ability to compensate. As a result, the kidney is fully compensated for PaCO210.6 kPa. The effect of acid poisoning on potassium metabolism: this type of acidosis is the same as in metabolic acidosis, which can be accompanied by hyperkalaemia. In particular, acute respiratory acid poisoning can be caused by the rapid decrease of pH, the increase of potassium, which can cause ventricular fibrillation in the heart, and death in cardiac arrest. The effect of hypercapnia: the increase in PaCO2 during respiratory acid poisoning, and the increase in plasma (H2CO3), known as hypercapnia (hypercapnia). It can cause vasomotor disturbance and neuropsychiatric disorders, mainly manifested in blood vessel congestion; The accumulation of CO2 can directly cause cerebral blood vessel dilation, the cerebral blood flow increases the brain congestion, the patient often has persistent headache, especially at night or early morning; Pulmonary encephalopathy. The effect of hypoxia: the accumulation of CO2 must be accompanied by hypoxia, so respiratory acid poisoning is often associated with hypoxia. 7, the change of some laboratory indexes PaCO2 increased significantly, is one of the characteristics of this type of acidosis, AB and CO2CP, AB because influenced by PaCO2 significantly increased, so the AB SB. Chronic respiratory acidosis after renal compensatory higher plasma [HCO - 3], therefore, SB and BB are increased, BE positive. Acute respiratory acidosis is normal for people who have no time to compensate for their kidneys Poor ventilation (1) airway obstruction, such as laryngeal edema, laryngospasm diphtheria, foreign bodies, obstruction, lymph nodes or tumor oppression windpipe, drowning, amnioti