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Chronic acquired demyelinating multiple neuropathy
1. Curr Neurol Neurosci Rep. 2014 Oct;14(10):487. doi: 10.1007/s11910-014-0487-z.
Neurologic manifestations of gastrointestinal and liver diseases.
Ferro JM(1), Oliveira S.
Author information:
(1)Department of Neurosciences, Service of Neurology, Hospital de Santa Maria,
University of Lisbon, Av. Prof. Egas Moniz, 1649-035, Lisboa, Portugal,
jmferro@fm.ul.pt.
Hepatic and gastrointestinal disorders can produce a wide spectrum of neurologic
complications both affecting the central nervous system (CNS) and the peripheral
nervous system. These manifestations range in severity from coma in acute liver
failure and acute pancreatitis, to minor cognitive changes in chronic
portosystemic encephalopathy and hepatitis C. Cerebrovascular diseases can
complicate hepatitis C infection and inflammatory bowel disease. Demyelinating
disorders may co-exist with inflammatory bowel disease. Anti-tumor necrosis
factor alpha drugs may induce demyelination. Ataxia may occur in malabsorption
syndromes and in gluten related disorders. Characteristic movement disorders are
key features of acquired hepatocerebral degeneration and of Whipple disease.
Multiple types of neuropathy can be found in association with hepatitis,
inflammatory bowel disease and gluten related disorders.
PMID: [PubMed - indexed for MEDLINE]
2. Neuroimaging Clin N Am. 2013 May;23(2):321-36. doi: 10.1016/j.nic.2012.12.010.
Epub 2013 Jan 23.
Mimics and rare presentations of pediatric demyelination.
OMahony J(1), Shroff M, Banwell B.
Author information:
(1)Divison of Neurology, Department of Pediatrics, Research Institute, The
Hospital for Sick Children, Toronto, Ontario, Canada.
This article reviews the features that should prompt consideration of diseases
that mimic acquired demyelinating syndromes and multiple sclerosis using
vignettes to highlight unusual clinical and radiologic features. Cases of
transverse myelitis, spinal infarction, acute disseminated
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