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贺银成西医综合(He Yincheng comprehensive Western Medicine).doc

贺银成西医综合(He Yincheng comprehensive Western Medicine).doc

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贺银成西医综合(He Yincheng comprehensive Western Medicine) 1, of 94 pages of some parts such as bronchial and bladder, although not by normal squamous epithelium lining, but can occur in squamous metaplasia. 2, page 12 hyalinization of renal tubular epithelial cells has a suction effect of vesicles, reabsorption of the urine protein, fuse with lysosomes to form hyaline bodies. Cytoplasm immunoglobulin quality rough endoplasmic reticulum protein accumulation, the formation of Rusell bodies, alcoholic liver disease liver cell deformation in the cytoplasm of keratin intermediate filaments before forming Mallory bodies. 3, page 16 common gangrene dry gangrene in artery occlusion but Hisamichi extremities venous smooth, wet gangrene occurred in the interconnected internal organs, such as lung, intestine, uterus, appendix, gallbladder, also occurred in the blocked artery occlusion and the vein of limbs. 4, 23 pages (complete regeneration of bone tissue regeneration) on cartilage regeneration (weak) muscle cells (weak) nerve cells (not) 5, hemorrhagic infarction 53 pages of intestine, lung and ovary 6, chemotaxis factor 69 page C5a LTB4, bacterial products, neutrophil cationic protein, IL-8 and TNF, IL-1 7, fibrinous inflammation 70 pages of lobar pneumonia, bacterial dysentery, diphtheria and villose heart 8, page 81 neuroblastoma as benign osteoblastoma, malignant neuroblastoma, medulloblastoma and nephroblastoma. Leukemia, seminoma and dysgerminoma are malignant tumors 9, TNM M is usually refers to distant metastasis, hematogenous metastasis 10, the body cell carcinoma metastasis rarely 11, primary granular contracted kidney in hypertension, secondary in chronic glomerulonephritis. AS can cause renal tissue infarction, infarction of left after the Great Depression scar. Chronic nephritis can cause renal papillary atrophy, renal scar number range, uneven distribution. 12, Aschoff cells formed by the 127 page macrophage proliferation aggregation fibrinoid necrosis after phagoc

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