原创力文档ve-statinegfl7 expression in endothelial cells is regulated by a distal enhancer and a proximal promoter under the direct control of erg and gata-2ve-statinegfl7表达内皮细胞是由一个远端增强器和一个近端启动子的直接控制下,erg和gata-2.pdfVIP
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ve-statinegfl7 expression in endothelial cells is regulated by a distal enhancer and a proximal promoter under the direct control of erg and gata-2ve-statinegfl7表达内皮细胞是由一个远端增强器和一个近端启动子的直接控制下,erg和gata-2
VE-statin/egfl7 Expression in Endothelial Cells Is
Regulated by a Distal Enhancer and a Proximal Promoter
under the Direct Control of Erg and GATA-2
Alexandra Le Bras1,2,3, Chantal Samson 1,2,3, Matteo Trentini1,2,3, Bertrand Caetano1,2,3, Etienne
1,2,3 1,2,3 4 1,2,3
Lelievre , Virginie Mattot , Friedrich Beermann , Fabrice Soncin *
´ ´
1 CNRS, Institut de Biologie de Lille, UMR 8161, Equipe labellisee La Ligue, Lille, France, 2 Universite Lille-Nord de France, Lille, France, 3 Institut Pasteur de Lille, F-59019
´ ´
Lille, France, 4 Swiss Institute for Experimental Cancer Research (ISREC), Centre de Phenotypage Genomique (CPG), School of Life Sciences, Swiss Federal Institute of
Technology (EPFL), Lausanne, Switzerland
Abstract
Angiogenesis is the process by which new blood vessels arise from existing ones by the budding out of endothelial cell
capillaries from the luminal side of blood vessels. Blood vessel formation is essential for organ development during
embryogenesis and is associated with several physiological and pathological processes, such as wound healing and tumor
development. The VE-statin/egfl7 gene is specifically expressed in endothelial cells during embryonic development and in
the adult. We studied here the regulatory mechanisms that control this tissue-specific expression. RT-qPCR analyses showed
that the specificity of expression of VE-statin/egfl7 in endothelial cells is not shared with its closest neighbor genes notch1
and agpat2 on the mouse chromosome 2. Chromatin-immunoprecipit
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