颞下颌关节骨病北卡罗莱纳州ppt课件.pptVIP

Craniomandibular Osteopathy Calvarial Hyperostosis;Craniomandibular Osteopathy (CMO);Etiology unknown Clinical signs seen at 3 – 8 months Mandibular swelling Difficulty prehending food Drooling Pain on opening of the mouth Physical exam- firm and often painful swelling of the mandible and/or the temporomandibular region; pyrexia may be noted Rarely involves long bone, but in terriers lameness or limb swelling may precede changes in the skull If severe = restricted jaw movements (if fusion between the tympanic bullae angular processes of the mandible) and masticatory muscle atrophy;Proliferative bone disease increased bone opacity Non-neoplastic Bones of endochondral origin = occipital bone, tympanic bullae and mandibular rami Bilateral but asymmetric irregular new bone of mandible and tympanic bulla-petrous temporal bone May see confinement to a specific region either mandible or tympanic bulla-petrous temporal area Calvarium and tentorium ossium are often thickened;Ettinger 6th ed. Volume 2; pg 1977;Diagnosis: signalment, clinical signs, radiographic or CT findings In atypical cases (rarely affected breeds or unilateral lesions) bone biopsy may be helpful Self-limiting – abnormal bone proliferation slows, becoming static at skeletal maturity (~1yr of age) Lesions may regress but radiographic abnormalities or prehensile dysfunction may remain Guarded prognosis if changes to affected areas are severe with ankylosis and adhesions that permanently restrict jaw movement ;CMOLiterature Review;VetRadUS, Vol. 35:2, 1994. pp 94-99;CMOLiterature Review;A syndrome of young male female Bullmastiffs 6 months of age Etiology unknown Benign hyperostosis of frontal and parietal bones – lesion localization is characteristic of the disease Other findings may include: Painful boney swelling of skull Lymphadenopathy Eosinophilia Pyrexia;Variant of CMO? Histologically calvarial hyperostosis is a primarily inflammatory subperiosteal or periosteal disease With CMO the inflammat