组会文章秦师兄.pptxVIP

Reporter: Qin Lei;Background ;Figure 7 Proposed mechanisms of the induction and maintenance of CD4t T-cell responses to respiratory syncytial virus (RSV) in wild-type or LC3b/ mice;Figure 1 LC3b/ mice develop increased lung pathology upon respiratory syncytial virus (RSV) infection.;Figure 2 Increased respiratory syncytial virus (RSV)-induced lung pathology in LC3b/ mice is interleukin (IL)-17a-dependent;Figure 3 Altered autophagic flux, innate cytokine production, and elicitation of CD4t T cell interleukin (IL)-17a production by LC3b/ dendritic cells (DCs) in response to respiratory syncyt