Clinical diagnosis and treatment of asymptomatic cerebral status.docVIP

Clinical diagnosis and treatment of asymptomatic cerebral status.doc

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Clinical diagnosis and treatment of asymptomatic cerebral status

 PAGE \* MERGEFORMAT 13 Clinical diagnosis and treatment of asymptomatic cerebral status Study: New Mengxin Ling Wang Yuqin Yuan Liu Asymptomatic cerebral infarction (silent cerebral infarction, SCI, also known as static cerebral infarction, is no clear clinical history and imaging studies of stroke infarct lesion or there is a history of stroke independent of infarction. Diagnosis of SCI mainly depends on CT, MRI examination [1], the elderly more common, such as SCI can not evolve into early intervention symptomatic cerebral infarction or development to vascular dementia (vascular dementia, VD) [2], and pseudobulbar palsy, Treatment is mainly directed against risk factors, control of blood pressure, lower blood sugar, blood lipids and blood viscosity, hypercoagulability of the blood to lift, thus further preventing multifocal cerebral pseudobulbar palsy due to the state and other complications. This review aims to draw the attention of scholars for further study. 1. Pathogenesis 1.1 Hemodynamics Systolic blood pressure is closely related with the level of SCI, the results showed that in patients with hypertension, those patients with significant postprandial hypotension often before the onset of symptoms in the nervous system of asymptomatic brain damage had been found, indicating that SCI may low blood pressure caused by brain perfusion on [3], and platelet aggregation of cerebral arterial lesions and enhanced the release of active substances is the key to the formation of cerebral infarction [4]. 1.2 CME Some scholars by transcranial Doppler scanning (TCD) emboli monitoring of the SCI were conducted, we found that high-intensity transient signals (HITS) as the representative of brain emboli are common, and cerebral microemboli proposed the concept of micro-embolism [5] . 1.3 abnormal glucose metabolism Clinical observation and animal experimental studies have shown that blood sugar levels and infarct size, brain edema

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