Power for the brain dysfunction of hippocampus of material progress.docVIP

Power for the brain dysfunction of hippocampus of material progress.doc

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Power for the brain dysfunction of hippocampus of material progress

 PAGE \* MERGEFORMAT 19 Power for the brain dysfunction of hippocampus of material progress [Abstract] At present, the power for the cerebral ischemia model of rat hippocampal cells in many reports on the relationship between the hippocampus and learning and memory research has become the focus of the study. In this paper, electrical brain dysfunction for physical effects of hippocampal briefly reviewed. [Keywords:] EA hippocampus substances The physiological function of the hippocampus is still explored. A large number of animal model studies have shown that the hippocampus related to learning and memory. When the hippocampus induced by cerebral ischemia and other damage, can cause a serious obstacle to learning and memory. Electric acupuncture points can be specified hippocampus of brain dysfunction Expression of substance. is for the current electrical brain dysfunction of hippocampal research material reviewed. 1 Power for the hippocampal cells the expression of apoptosis-related protein 1.1 Bcl 2 is the most feature-specific antagonist of apoptosis protein Bcl 2 gene for the extension of the basic biological function of cell life span, increase the cells resistance to a variety of apoptotic stimuli. Bax and Bcl 2 role in contrast, can contribute to wither apoptosis, Bcl 2 expression level is high, the formation of Bcl 2/Bcl 2 homodimer, inhibition of apoptosis, Bax expression levels, the formation of Bax / Bax homodimers, accelerated apoptosis, Bcl 2 and Bax levels were equal, the heterodimer formation of Bcl 2/Bax termination of apoptosis. Recent research suggests that Bcl 2 and Bax regulation of apoptosis, the level of expression depends not only on their own, but also with the Bax / Bcl 2 ratio, and when the ratio increases, the cells tend to apoptosis [1]. caspase 3 activation is a key trigger of apoptosis (a “molecular switch,” said), is the final execution of apoptosis protein. Bc1 2 gene family in the regulation of mitochondrial perme

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