Synergistic induction of macrophage inflammatory Protein-3αCCL20 production by interleukin-17A and tumor necrosis factor-α in nasal polyp fibroblasts 英文参考文献.docVIP

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Synergistic induction of macrophage inflammatory Protein-3αCCL20 production by interleukin-17A and tumor necrosis factor-α in nasal polyp fibroblasts 英文参考文献.doc

Synergistic induction of macrophage inflammatory Protein-3αCCL20 production by interleukin-17A and tumor necrosis factor-α in nasal polyp fibroblasts 英文参考文献

ORIGINALARTICLE SynergisticInductionofMacrophageInflammatory Protein-3?/CCL20ProductionbyInterleukin-17Aand TumorNecrosisFactor-?inNasalPolypFibroblasts ManabuNonaka,MD,PhD,NozomuOgihara,MD,AkiraFukumoto,MD,PhD, AtsukoSakanushi,MD,KaoruKusama,MD,RubyPawankar,MD,PhD, andToshiakiYagi,MD,PhD features that are similar to those in patients with chronic Background: Accumulation of T cells and immature dendritic sinusitis. Many of the features of the chronic sinusitis cells (DCs) is one of the characteristic features of nasal polyps. environment, such as the presence of T and B cells, However, the

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