喂食康敏元中唾液乳杆菌抑制过敏呼吸道反应动物实验结论(The results of the study on the control of salivary lactobacillus in the feeding of kangmin were concluded).docVIP
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喂食康敏元中唾液乳杆菌抑制过敏呼吸道反应动物实验结论(The results of the study on the control of salivary lactobacillus in the feeding of kangmin were concluded)
喂食康敏元中唾液乳杆菌抑制过敏呼吸道反应动物实验结论(The results of the study on the control of salivary lactobacillus in the feeding of kangmin were concluded)
The results of the study were carried out in response to the experimental results of the respiratory tract reaction in the respiratory tract
Oral administration of Lactobacillus salivarius
Inhibits the allergic airway response in mice
Chia-yang Li, hung-chih Lin, Kai - Chung Hsueh, shu-fen Wu, and shih-hua
Fang
In this paper,
Asthma is one of the internationally recognized chronic inflammatory diseases of the respiratory tract. In this study, the effects of probiotics were further studied in the antibody stimulation test (OVA), which caused the asthma of BALB/c mice. In antigen stimulation test before giving mice given l. salivarius PM - A0006, respectively analysis: after the stimulation test OVA antibody concentration in the blood and the respiratory tract of acetyl choline reactions, lung inflammation cells, bronchoalveolar lavage and cytokines in the spleen. The results showed that l. salivarius pm-a0006 of feeding mice significantly reduced the concentration of eosinophilic white blood cells in the respiratory tract and reduced the amount of ov-specific antibody in the blood. In addition, L. Salivarius pm-a0006 can also reduce the incidence of respiratory allergic reactions and increase IFN - gamma content.
According to the results, edible l. salivarius pm-a0006 probiotics has therapeutic effects on allergic respiratory diseases.
preface
Asthma is a chronic airway disease characterized by reversible airway inflammation, obstruction, overreaction, and increased IgE production and mucus production. Inflammatory cells, such as eosinophilic granulocytes, lymphocytes and mast cells, which are associated with bronchial infiltration, reflect the severity of asthma. Several inflammatory mediators promote the development of asthma, such as PGE2, LTC4, and chemokine. Previous studies have shown that Th2 cells and their cytokines play an i
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