原创力文档a combined cxcl10, cxcl8 and h-fabp panel for the staging of human african trypanosomiasis patients结合cxcl10 cxcl8和研究小组举办非洲人类锥虫病的患者.pdfVIP
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a combined cxcl10, cxcl8 and h-fabp panel for the staging of human african trypanosomiasis patients结合cxcl10 cxcl8和研究小组举办非洲人类锥虫病的患者
A Combined CXCL10, CXCL8 and H-FABP Panel for the
Staging of Human African Trypanosomiasis Patients
1 1 1 2 ´ 3
Alexandre Hainard , Natalia Tiberti , Xavier Robin , Veerle Lejon , Dieudonne Mumba Ngoyi , Enock
4 5 1 6 ´ ´ 7
Matovu , John Charles Enyaru , Catherine Fouda , Joseph Mathu Ndung’u , Frederique Lisacek ,
¨ 7 1 1
Markus Muller , Natacha Turck , Jean-Charles Sanchez *
1 Biomedical Proteomics Research Group, Medical University Centre, Geneva, Switzerland, 2 Department of Parasitology, Institute of Tropical Medicine, Antwerp, Belgium,
3 Institut National de Recherche Biomedicale, Kinshasa, D.R. Congo, 4 Department of Veterinary Parasitology and Microbiology, Faculty of Science, Makerere University,
Kampala, Uganda, 5 Department of Biochemistry, Faculty of Science, Makerere University, Kampala, Uganda, 6 Foundation for Innovative New Diagnostics (FIND), Geneva,
Switzerland, 7 Swiss Institute of Bioinformatics, Medical University Centre, Geneva, Switzerland
Abstract
Background: Human African trypanosomiasis (HAT), also known as sleeping sickness, is a parasitic tropical disease. It
progresses from the first, haemolymphatic stage to a neurological second stage due to invasion of parasites into the central
nervous system (CNS). As treatment depends on the stage of disease, there is a critical need for tools that efficiently
discriminate the two stages of HAT. We hypothesized that markers of brain damage discovered by proteomic strategies and
inflammation-related proteins could individually or in combination indi
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