非st段抬高性急性冠状动脉综合征2009（Non ST segment elevation acute coronary syndrome 2009）.docVIP

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非st段抬高性急性冠状动脉综合征2009（Non ST segment elevation acute coronary syndrome 2009）.doc

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非st段抬高性急性冠状动脉综合征2009（Non ST segment elevation acute coronary syndrome 2009）

非st段抬高性急性冠状动脉综合征2009（Non ST segment elevation acute coronary syndrome 2009） Guangdong General Hospital Author: Jin Lijun Non ST segment elevation acute coronary syndromes (NSTE-ACS) include unstable angina pectoris (UAP) and non ST segment elevation myocardial infarction (NSTEMI), formerly referred to as non Q Wave myocardial infarction (NQMI), is caused by the instability of atherosclerotic plaques, related artery incomplete occlusive thrombus in platelet based white thrombus, different from the acute coronary ST elevation syndrome (STE-ACS) of coronary artery occlusion red thrombus. Therefore, the therapeutic strategy of NSTE-ACS differs greatly from that of STE-ACS. Epidemiology and pathophysiology In the United States, more than 1 million 400 thousand people are admitted to ACS for [1] each year, 70% of whom have been diagnosed with UAP and NSTEMI, and 30% with ST elevation myocardial infarction (STEMI). According to a global registry, the six-month survival rate of NSTE-ACS patients improved from 1999 to 2006. The United States each year because of the number of deaths from coronary heart disease by about half control due to the risk factors such as lowering serum cholesterol, effectively control blood pressure, smoking, mostly thanks to the treatment of improved [2 and 3]. In addition, also benefited from lower detection of cardiac specific troponin the mortality of patients with clinical myocardial necrosis, the more sensitive (and can identify a smaller range of NSTEMIs), now has as a diagnosis of myocardial necrosis troponin specific biomarkers. Over the past year or so, pathophysiological studies have led to a better understanding of coronary plaque rupture, providing a pathophysiological basis for most of the ACS events, [4]. As we can better understand the biological behavior of coronary plaques, we hope that we will be able to better identify vulnerable lesions and find more effective ways to prevent, diagnose and treat coronary artery disease. Treatmen