心电图--培训PPT.ppt

Sino-Atrial Exit Block Atrio-Ventricular (AV) Block ? 1st Degree AV Block  ?Type I (Wenckebach) 2nd Degree AV Block  ?Type II (Mobitz) 2nd Degree AV Block  ?Complete (3rd Degree) AV Block  ?AV Dissociation Intraventricular Blocks ?Right Bundle Branch Block  ?Left Bundle Branch Block ?Left Anterior Fascicular Block  ?Left Posterior Fascicular Block  ?Bifascicular Blocks  ?Nonspecific Intraventricular Block ?Wolff-Parkinson-White Preexcitation  Right Bundle Branch Block ?Left Bundle Branch Block ?Left Anterior Fascicular Block ?Wolff-Parkinson-White Preexcitation Left Ventricular Hypertrophy (LVH) Myocardial Infarction ?A. Normal ECG prior to MI  B. Hyperacute T wave changes - increased T wave amplitude and width; may also see ST elevation ?C. Marked ST elevation with hyperacute T wave changes (transmural injury) ?D. Pathologic Q waves, less ST elevation, terminal T wave inversion (necrosis) ? ?E. Pathologic Q waves, T wave inversion (necrosis and fibrosis)  ?F. Pathologic Q waves, upright T waves (fibrosis) The normal U Wave: (the most neglected of the ECG waveforms) ?U wave amplitude is usually 1/3 T wave amplitude in same lead  ?U wave direction is the same as T wave direction in that lead  U waves are more prominent at slow heart rates and usually best seen in the right precordial leads.  ?Origin of the U wave is thought to be related to afterdepolarizations which interrupt or follow repolarization.  ECG Rhythm Abnormalities  Supraventricular arrhythmias Ventricular arrhythmias Premature atrial complexes Premature junctional complexes Atrial fibrillation Atrial flutter Paroxysmal supraventricular tachycardia Ectopic atrial tachycardia and rythm Premature Ventricular Complexes (PVCs) Ventricular Tachycardia Ventricular Fibrillation ECG Conduction Abnormalities  ?Atrio-Ventricular (AV) Block ?S