肝胆疾病研究团.doc免费

肝胆疾病研究团队 一、团队负责人 代荣阳 博士，教授，硕士生导师，澳门科技大学兼职博士生导师，哈佛医学院访问学者，教育部新世纪优秀人才支持计划获得者，四川省学术技术带头人后备人选。 二、团队主要人员 代荣阳博士； 赵晓芳博士； 周虹博士； 成鹰博士； 姚富丽博士； 李玉筱博士； 甘淋博士； 刘秋均博士； 等。 三、研究方向 研究团队致力于肝胆疾病细胞信号调控研究，目前主要从事应激反应与肝胆疾病的关系研究。 四、主要论文（*，通讯作者；+并列第一） [01]Feng C, He K, Zhang C, Su S, Li B, Li Y, Duan CY, Chen S, Chen R, Liu Y, Li H, Wei M, Xia X, Dai R*. JNK Contributes to the Tumorigenic Potential of Human Cholangiocarcinoma Cells through the mTOR Pathway Regulated GRP78 Induction. PLoS One. 2014;9(2):e90388. [02]RY Dai, XF Zhao, JJ Li, R Chen, ZL Luo, LX Yu, SK Chen, CY Zhang, CY Duan, YP Liu, CH Feng, XM Xia, H Li, J Fu, HY Wang*. Implication of transcriptional repression in compound C-induced apoptosis in cancer cells. Cell Death Dis. 2013;4:e883. [03]Chen R, Duan CY, Chen SK, Zhang CY, He T, Li H, Liu YP, Dai RY*. The suppressive role of p38 MAPK in cellular vacuole formation. J Cell Biochem. 2013;114:1789-99. [04]Chen R, Li J, Feng C, Chen S, Liu Y, Duan C, Li H, Xia X, He T, Wei M, Dai R*. c-Met function requires N-linked glycosylation modification of pro-Met. J Cell Biochem. 2013;114(4):816-22. [05]Dai R, Li J, Fu J, Chen Y, Wang R, Zhao X, Luo T, Zhu J, Ren Y, Cao J, Qian Y, Li N, Wang H*. The Tyrosine Kinase c-Met Contributes to the Pro-tumorigenic Function of the p38 Kinase in Human Bile Duct Cholangiocarcinoma Cells. J Biol Chem. 2012;287(47):39812-23. [06]Dai R, Li J, Fu J, Chen Y, Yu L, Zhao X, Qian Y, Zhang H, Chen H, Ren Y, Su B, Luo T, Zhu J, Wang H*. Disturbance of Ca2+ homeostasis converts pro-Met into non-canonical tyrosine kinase p190MetNC in response to endoplasmic reticulum stress in MHCC97 cells. J Biol Chem. 2012;287(18):14586-97. [07]Dai R, Yan D, Li J, Chen S, Liu Y, Chen R, Duan C, Wei M, Li H*, He T. Activation of PKR/eIF2α signaling cascade is associated with dihydrotestosterone-induced cell cycle arrest and apoptosis in human liver cells. J Cell Biochem. 2012;113(5):1800-8. [08]Run Chen, Rongyang Dai +, Chunyan Duan, Youping Liu, Shaokun Chen, Dongmei Y