原创力文档crosstalks between myo-inositol metabolism, programmed cell death and basal immunity in arabidopsis肌醇代谢之间的串扰,程序性细胞死亡和基底在拟南芥免疫力.pdfVIP
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crosstalks between myo-inositol metabolism, programmed cell death and basal immunity in arabidopsis肌醇代谢之间的串扰,程序性细胞死亡和基底在拟南芥免疫力
Crosstalks between Myo-Inositol Metabolism,
Programmed Cell Death and Basal Immunity in
Arabidopsis
1.¤ ´ 1. 2 1 1
Ping Hong Meng , Cecile Raynaud *, Guillaume Tcherkez , Sophie Blanchet , Kamal Massoud ,
´ 1 1 3 2
Severine Domenichini , Yves Henry , Ludivine Soubigou-Taconnat , Caroline Lelarge-Trouverie ,
1 3 1
Patrick Saindrenan , Jean Pierre Renou , Catherine Bergounioux
´ ˆ ´ ´
1 Institut de Biotechnologie des Plantes, UMR CNRS 8618, Universite Paris-Sud XI, bat 630, Plateau de Moulon, Orsay, France, 2 Plateforme Metabolisme Metabolome
´ ˆ ´ ´ ´ ´
IFR87, Institut de Biotechnologie des Plantes, Universite Paris-Sud XI, bat 630, Plateau du Moulon, Orsay, France, 3 Unite de Recherche en Genomique Vegetale, 2, CP5708,
Evry, France
Abstract
Background: Although it is a crucial cellular process required for both normal development and to face stress conditions,
the control of programmed cell death in plants is not fully understood. We previously reported the isolation of ATXR5 and
ATXR6, two PCNA-binding proteins that could be involved in the regulation of cell cycle or cell death. A yeast two-hybrid
screen using ATXR5 as bait captured AtIPS1, an enzyme which catalyses the committed step
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