Preservation of the Blood Brain Barrier and Cortical Neuronal Tissue by Liraglutide, a Long Acting Glucagon-Like-1 Analogue, after Experimental Traumatic Brain Injury(血脑屏障的皮层神经组织的保护作用glucagon-like-1利拉鲁肽,长时间模拟,创伤性脑损伤).PDFVIP
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RESEARCH ARTICLE
Preservation of the Blood Brain Barrier and
Cortical Neuronal Tissue by Liraglutide, a
Long Acting Glucagon-Like-1 Analogue, after
Experimental Traumatic Brain Injury
1,2 1 2,3† 1,3
Jakob Hakon *, Karsten Ruscher , Bertil Romner , Gregor Tomasevic
1 Laboratory for Experimental Brain Research, Wallenberg Neuroscience Center, Lund University, BMC
A13, Lund, Sweden, 2 Department of Neurosurgery, Copenhagen University Hospital, Risgshospitalet,
Denmark, 3 Department of Neurosurgery, University Hospital of Lund, Lund, Sweden
† Deceased.
* jakobhakon@
a11111
Abstract
Cerebral edema is a common complication following moderate and severe traumatic brain
injury (TBI), and a significant risk factor for development of neuronal death and deterioration
of neurological outcome. To this date, medical approaches that effectively alleviate cerebral
OPEN ACCESS edema and neuronal death after TBI are not available. Glucagon-like peptide-1 (GLP-1) has
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