Abomasal Displacement - All课件.pptVIP

Abomasum Barb Knust Outline: Displaced Abomasum History and Signalment Pathophysiology Diagnosis Clinical signs, clin path, R/O’s Treatment Non-surgical Surgical (4 approaches) Ancillary care (Fluids, Abx, …) Risk Factors for LDA Displaced Abomasums DA’s, LDA’s, RDA’s, RTA’s Adult lactating dairy Production problem Herd problem [related to nutrition] Majority of DA’s have concurrent diseases History and Signalment of DA Age: older lactating dairy cattle Timing: 80% occur during first month after parturition Nutrition: Dry cow rations: +DCAD / inadeq efv fiber Fresh cow: excess NSC’s / inadeq efv fiber Normal location of abomasum Left view bovine stomach Why does the abomasum displace? (1) Abomasal atony (2) Increased abomasal gas production (1) + (2) = abomasum moves (LDA,RDA) Displacing Abomasum In Action LDA Why does abomasal atony occur? Hypocalcaemia due to +DCAD, [Ca]blood, mastitis, - E balance 7 times more likely to develop DA’s Inadequate effective fiber VFA’s reach abomasum = abomasal hypomotility = HCl refluxes back into rumen = systemic metabolic alkalosis Endotoxemia Released during Gm – sepsis (mastitis/metritis) Why increased gas productn? NSC : effective fiber ratio Clinical Pathology Normal CBC Metabolic alkalosis(slight) Hypo Ca K Cl Ketosis (mild) Dehydration Hypoglycemia (maybe) Hyperbilirubinemia Clinical Signs of DA’s Normal TPR (most cases) Partial anorexia (“off feed”) Hypogalactia (“down in milk” ~ 5-10 lb/day) Depression (ADR) Secondary ketosis mild to moderate Scant stool firm/loose undigested particles Clinical Signs (continued) Paralumbar fossa: “slab-sided” abdomen Visualize / Palpate PLF Rectal palpation (can’t) Mild colic Mild hypocalcemia Hypotonic rumen Cold ears, widely dilated pupils Clinical Signs (continued) LDA: Ping Splash Ascult and percuss Ping high pitched Ballottment for splash of fluid All pings are not created equal – rumen ping Differential Diagnosis LDA R/O’s 1° ketosis (non-pinging LDA) Rumen ping RD