子宫内膜CD82和S100A10%2fAnnexin+A2的表达调控及其和内膜接受态相关性研究.pdfVIP

n1i TOOOOOOOOOOOOO0600000D………………………………………………28O…0OOO………………………………10…………29 BDDOOOOOOOOOOOOOOOOOOOOOOOOOOOOOOOO00·Oooooo0oD………………………………73O…………52O………………………………………66o………………………………58 n100A1i 2 FUT432．Annexin withA2Molecularmatrix．CD82Weimoleculesmetastasis．Decidualmatemal—fetalmarkermaintenancemembranesuterineYanbetweennotTheTherehasbeen4aaaareaasaareaRU486onLiuatactsCanTheadhesionadhesionandadhesionandCD82ofofsomeotherofofofendometrialandoftototofacilitatesforandShuaiandandcellscellsofthetransmembranethethatcommontheendometriumthethetheextracellularthetheendometriumthethetransfersseriesestablishmentendometrialsitesIVwhichisinvolvedisinhibitinintoinmiceinterfaceisinendometrialitisinvolvedendometriumCD82cellandofmoleculesandisofcellsrhesusinin A2 in of increases duringgestation monkeysearlypregnancy．Annexinprogressively humanfetomaternalinterface． Inthis researchthe and of 100A10and CD82，S study，we expressionregulation theirroles AnnexinA2oncell evaluate oncellsurface surface，and duringimplantation． ofthemechanismin The will thefurtherelucidation embryoimplantaion． studypromte II．Methods 100A10andAnnexinA2inthehuman andlocalizationofCD82．S 1．Expression endometriumwere byimmunohistochemistry． analysed wastransfectedintocells． 2．Therecombinantvector FUT4weredetectedthemethodsofRT—PCR． 3．The 0fCD82and expression by