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PAGE \* MERGEFORMAT 8
Inflammation of the liver disease on the CT diagnosis
[Keywords:] CT diagnosis of inflammatory disease of the liver (A) of the liver abscess Amebic abscess amebic trophozoites by dissolving tissue lesions by intestinal blood flow into the portal vein system and then into the liver. Intrahepatic portal vein obstruction due to parasite peripheral support, and produce soluble tissue enzymes, resulting in liver ischemia , necrosis, followed by the formation of abscesses. mostly secondary to amebic colitis.
The formation and development of liver abscess: inflammation of the liver abscess in front of a process of bacterial abscesses were formed about a week or so. Start small abscess about the size of several millimeters, National Cheng Kung University, after merging abscess, up to several centimeters in size. and some start is the large abscess. Abscess center filled with necrotic tissue by white blood cells and the formation of semi-liquid residue, course development, internal cell components decreased, the contents tend to liquefaction. Usually unilocular, or multilocular with the question of the wall, in the repair process, vasodilation, cell proliferation, proliferation of fibrous connective tissue and the formation of connective tissue rich in blood vessels of the capsule.
The development of amebic were a little slow, and amebic colitis in about 30 to 40 days after formation. Because of parasite reproduction within the portal vein obstruction, resulting in infarction of liver cells, and soluble tissue enzymes direct damage to liver cells and the formation of abscess. Vomica generally higher, often secondary bacterial infections.
Abscess, perforation, and the surrounding tissue or organs fistula formation, up to penetrate the diaphragm to form a subphrenic abscess, empyema formation of penetration of the chest; violations of the formation of lung abscess, and bronchial same, then the formation of bronchial
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