Rapid aging of learning and memory defects in mice and the biochemical mechanisms.docVIP

 PAGE \* MERGEFORMAT 23 Rapid aging of learning and memory defects in mice and the biochemical mechanisms [Abstract] Objective of senescence accelerated mouse (SAMP8) simulation of Alzheimer’s, to observe the learning and memory ability and learning and memory ability of the possible mechanisms. Methods 10 months old SAMP8, and with the age of senescence accelerated mouse resistant (SAMR1) as normal controls. water lost memory and spatial learning test in recent memory. detected by biochemical mitochondrial membrane potential and ATP cortical activity observed mitochondrial function, d