pbl资料（PBL data）.docVIP

pbl资料（PBL data） Cerebral infarction treatment: * lowering blood pressure and intracranial pressure If the systolic pressure exceeds 220mmHg or diastolic pressure exceeds 120mmHg, every 15 minutes to retest, and three times all the same, should be sublingual Kato Pury (captopril) to slow down about 15% of the blood pressure. Rapid intravenous drip of mannitol, 20 to 100g per 2~6 hours. For lowering intracranial pressure drugs are: albumin, glycerol saline, furosemide etc.. (I) thrombolytic therapy: within 3~6 hours after onset. Intravenous thrombolysis, intra-arterial thrombolysis, and intra-arterial thrombolysis have not been widely used in clinical practice. Commonly used drugs, urokinase plasminogen activator (t-PA). The main risk and side effect of thrombolytic therapy is intracranial hemorrhage, and the chance of cardiogenic embolism and cerebral hemorrhage is higher. No anticoagulant or aspirin should be used within the first 24 hours after treatment. 24 hours later, CT showed no bleeding, and feasible antiplatelet and / or anticoagulant treatment (two) anticoagulant therapy: the common drugs are heparin, low molecular heparin, and must be tested for blood coagulation. The main side effect is bleeding, in which low molecular weight heparin is safer than unfractionated heparin. (three) anti platelet drugs: (1) aspirin is economical, practical, safe and the most routine antiplatelet prophylaxis. The lowest effective dose is 50mg or 75mg / day. The acute phase can be increased by dosage to 300mg / day. The use of the medication does not require a blood test. Aspirin can significantly reduce side effects. (2) for the treatment of ticlopidine, medication and preventive medication, dosage and usage is 125 ~ 250mg / day, oral meal. Blood, liver function, blood coagulation and so on should be detected during the course of administration. A small number of patients may suffer from neutropenia, jaundice and elevation of transaminase, side effects of bleeding, prolonged b