the colocalization potential of hiv-specific cd8+ and cd4+ t-cells is mediated by integrin β7 but not ccr6 and regulated by retinoic acid的colocalization潜力hiv-specific cd8 +及cd4 + t细胞是由整合素β7但不是ccr6和由视黄酸.pdfVIP
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thecolocalizationpotentialofhiv-specificcd8andcd4t-cellsismediatedbyintegrinβ7butnotccr6andregulatedbyretinoicacid的colocalization潜力hiv-specificcd8及cd4t细胞是由整合素β7但不是ccr6和由视黄酸
The Colocalization Potential of HIV-Specific CD8+ and
CD4+ T-Cells is Mediated by Integrin b7 but Not CCR6 and
Regulated by Retinoic Acid
1,2 3 2 1,2,4 1
Vanessa Sue Wacleche , Nicolas Chomont , Annie Gosselin , Patricia Monteiro , Mathieu Goupil ,
1,2 ´ 1,2 5 6
Hassen Kared , Cecile Tremblay , Nicole Bernard , Mohamed-Rachid Boulassel , Jean-
Pierre Routy4,6,7, Petronela Ancuta1,2,4*
´ ´ ´ ´
1 Department of Microbiology and Immunology, Universite de Montreal, Montreal, Quebec, Canada, 2 Centre Hospitalier de l’Universite de Montreal (CHUM)-Research
´
Center, Saint-Luc Hospital, Montreal, Quebec, Canada, 3 VGTI-Florida, Port St Lucie, Florida, United States of America, 4 INSERM Unit 743, Montreal, Quebec, Canada,
5 Research Institute of the McGill University Health Centre, Montreal, Quebec, Canada, 6 Division of Hematology, McGill University Health Centre, Montreal, Quebec,
Canada, 7 Immunodeficiency Service, Montreal Chest Institute, McGill University Health Centre, Montreal, Quebec, Canada
Abstract
CD4+ T-cells from gut-associated lymphoid tissues (GALT) are major targets for HIV-1 infection. Recruitment of excess
effector CD8+ T-cells in the proximity of target cells is critical for the control of viral replication. He
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