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Cardiometabolic Risk: Evaluation Treatment in Your Patient Population;Why Focus on Cardiometabolic Risk? ;Cardiometabolic Risk;The State of Risk;Direct and Indirect Cost of CVD and Diabetes;Abnormal Lipid Metabolism LDL ? ApoB ? HDL ? Trigly. ? ;Non-modifiable;Case - Mr. Martin;Cardiovascular Risk Factor Trends Among U.S. Adults Aged 20-74;Centers for Disease Control and Prevention. National diabetes fact sheet: general information and national estimates on diabetes in the United States, 2005. Atlanta, GA: U.S. Department of Health and Human Services, Centers for Disease Control and Prevention, 2005.; Insulin Resistance ;Factors affectinginsulin resistance;IFG and IGT;Interpreting BloodGlucose Levels;Criteria for testing for type 2 diabetesin asymptomatic children50;Criteria for testing for diabetes in asymptomatic adult individuals50;Criteria for testing for diabetes in asymptomatic adult individuals50;Criteria for testing for diabetes in asymptomatic adult individuals50;0;Insulin Sensitivity;Overweight/Obesity;Understanding Cardiometabolic Risk: Broadening Risk Assessment and Management;Screening: Overweight;Measuring Waist Circumference;Primary Metabolic Disturbance;Body Weight and CVD;Risk ManagementOverweight ;Risk Management, cont. Overweight ;Abnormal Lipid Metabolism;Total Cholesterol Goals34;Abnormal Lipid Metabolism;Major Risk FactorsAffecting Lipid Goals36;Statins (also called HMG-CoA reductase inhibitors) work by increasing hepatic LDL-C removal from the blood. Resins (also called bile acid sequestrants) bind to bile acids in the intestines and prevent their reabsorption, leading to increased hepatic LDL-C removal from the blood. Cholesterol absorption inhibitors help lower LDL-C by reducing the amount of cholesterol absorbed in the intestines; increases LDL receptor activity. ;Fibrates (also called fibric acid derivatives) activate an enzyme that speeds the breakdown of triglyceriderich lipoproteins while also increasing HDL-C. Niacin