Postprandial lipid metabolism in high fat-related disease research and progress.docVIP

 PAGE \* MERGEFORMAT 14 Postprandial lipid metabolism in high fat-related disease research and progress [Abstract] hyperlipidemia are all causes blood cholesterol and triglycerides and related proteins increased. Hyperlipidemia is a serious public health problem. Over the years, many clinical and research focus on fasting changes in blood lipid levels, while ignoring the human body in 24 hours in most of the time in the postprandial lipid dynamic changes. postprandial dyslipidemia of atherosclerosis is an important factor in the development. So of postprandial lipid metabolism anomaly and its related diseases is a major feature in recent years. In this paper, research progress in this area are reviewed. [Key Words] high-fat meal lipid metabolism-related diseases Lipid parameters as atherosclerosis (AS predictor of the risk factors have been identified in epidemiology, and has been widely used in clinical and scientific research, lipid parameters currently used are determined by the results of the fasting state, but Most of the time in human postprandial (food digestion and the subsequent 6 ~ 8h in the state, therefore, fasting lipid parameters is difficult to state accurately reflect the process of lipid metabolism in the body, and with the determination of postprandial lipid parameters more accurately reflect state of lipid metabolism [1]. of abnormal postprandial lipid metabolism and its related diseases is a major feature in recent years. In this paper, research progress in this area are reviewed. 1 and postprandial hyperlipidemia postprandial state Postprandial state is the food to blood glucose, amino acids and triglycerides and other nutrients back to the meal This period of time before the level of physiological and pathological changes. Normally, the postprandial metabolic state lasted only 1 hour after a meal [2] with reference to domestic research results to determine the serum TG levels after fat meal 4h (TG4hgt; 2mmo1 / L