Oral phosphatidylcholine pretreatment alleviates the signs of experimental rheumatoid arthritis 英文参考文献.docVIP
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Oral phosphatidylcholine pretreatment alleviates the signs of experimental rheumatoid arthritis 英文参考文献
Available online /content/11/2/R43
Research article
Open Access
Vol 11 No 2
Oral phosphatidylcholine pretreatment alleviates the signs of
experimental rheumatoid arthritis
Gabor Er?s1,2, Saleh Ibrahim3, Nikolai Siebert1, Mihály Boros2 and Brigitte Vollmar1
1Institute for Experimental Surgery, University of Rostock, Schillingallee 69a, Rostock D-18057, Germany
2Institute of Surgical Research, University of Szeged, Pécsi u. 6, Szeged H-6720, Hungary
3Immunogenetics Group, University of Rostock, Schillingallee 70, Rostock D-18057, Germany
Corresponding author: Brigitte Vollmar, brigitte.vollmar@med.uni-rostock.de
Received: 2 Dec 2008 Revisions requested: 18 Jan 2009 Revisions received: 16 Feb 2009 Accepted: 18 Mar 2009 Published: 18 Mar 2009
Arthritis Research Therapy 2009, 11:R43 (doi:10.1186/ar2651)
This article is online at: /content/11/2/R43
? 2009 Er?s et al.; licensee BioMed Central Ltd.
This is an open access article distributed under the terms of the Creative Commons Attribution License (/licenses/by/2.0),
which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited.
Abstract
Introduction Phosphatidylcholine and phosphatidylcholine-
derived metabolites exhibit anti-inflammatory properties in
various stress conditions. We hypothesized that dietary
were determined, and classical histological analysis was
performed.
Results
collagen-induced
Phosphatidylcholine
arthritis-induced
pretreatment
hypersensitivity,
reduced
the
and
phosphatidylcholine
may
potentially
function as an anti-
decrease inflammatory
inflammatory
substance
and
may
decreased the number of leukocyte–endothelial cell interactions
and the extent of functional capillary density as compared with
those of group 1. It also ameliorated the tissue damage and
decreased inducible nitric oxide synthase expression. The
expressions of the cannabinoid receptors and TNFα were
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