Short-term pacing in the mouse alters cardiac expression of connexin43 英文参考文献.docVIP

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Short-term pacing in the mouse alters cardiac expression of connexin43 英文参考文献.doc

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Short-term pacing in the mouse alters cardiac expression of connexin43 英文参考文献

BMC Physiology BioMedCentral Research article Open Access Short-term pacing in the mouse alters cardiac expression of connexin43 Andrianos Kontogeorgis1,4, Riyaz A Kaba3,4, Eunice Kang1, Jonathan E Feig1, Pritha P Gupta1, Marc Ponzio1, Fangyu Liu1, Michael J Rindler2, Andrew L Wit3, Edward A Fisher1,2, Nicholas S Peters3,4 and David E Gutstein*1,2 Address: 1Leon H. Charney Division of Cardiology, Department of Medicine, New York University School of Medicine, New York, NY, USA, 2Department of Cell Biology, New York University School of Medicine, New York, NY, USA, 3Department of Pharmacology, Columbia University College of Physicians and Surgeons, New York, NY, USA and 4Department of Cardiology, St Marys Hospital, Imperial College London, UK Email: Andrianos Kontogeorgis - a.kontogeorgis@imperial.ac.uk; Riyaz A Kaba - r.kaba@imperial.ac.uk; Eunice Kang - eyk216@; Jonathan E Feig - feigj01@; Pritha P Gupta - pg769@; Marc Ponzio - ponzim01@; Fangyu Liu - liuf02@; Michael J Rindler - rindlm01@; Andrew L Wit - alw4@; Edward A Fisher - Edward.Fisher@; Nicholas S Peters - n.peters@imperial.ac.uk; David E Gutstein* - david.gutstein@ * Corresponding author Published: 6 May 2008 Received: 12 December 2007 Accepted: 6 May 2008 BMC Physiology 2008, 8:8 doi:10.1186/1472-6793-8-8 This article is available from: /1472-6793/8/8 ? 2008 Kontogeorgis et al; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Abstract Background: Cardiac insults such as ischemia, infarction, hypertrophy and dilatation are often accompanied by altered abundance and/or localization of the connexin43 gap junction protein, which may predispose towards arrhythmic complications. Models of chronic dyssynchronous cardiac activation have also