瘫痪的定位诊断（Localization diagnosis of paralysis）.docVIP

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瘫痪的定位诊断（Localization diagnosis of paralysis）.doc

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瘫痪的定位诊断（Localization diagnosis of paralysis）

瘫痪的定位诊断（Localization diagnosis of paralysis） Localization diagnosis of paralysis Paralysis is the inability of the muscles to contract completely. According to the degree of disability, it can be divided into two types, incomplete paralysis (paresis, muscle strength examination for 1-4 degrees) and complete paralysis (muscle strength is 0 degrees). There are three causes of paralysis: (I) neurogenic paralysis: according to the damaged parts of the motor pathway, it is divided into: 1. motor neuron paralysis: paralysis resulting from lesions of the pyramidal tract in the cortical motor area to the anterior horn of the spinal cord. The characteristics are: (1) the scope of paralysis is extensive. Due to the release of stretch reflex after pyramidal tract injury, the upper limb flexors and extensor muscles of the extensor muscles of the paralyzed limb are increased, which is called spastic paralysis. But in the acute phase (shock) hypotonia, showed flaccid paralysis. The normally inhibited tendon reflex is released and tendon jerk occurs. The primitive reflex, which is normally suppressed, appears again, that is, the pathological reflex is positive. In addition to the paralysis after a long illness in disuse atrophy, no muscle atrophy. No reaction of degeneration 6. Detection. Paralysis caused by lesion of cortical motor area, although paralyzed in motor neuron, is different in clinical manifestations (see later). 2. paralysis of motor neuron: paralysis caused by damage of anterior horn, anterior root, nerve plexus and peripheral nerve of the spinal cord. Its characteristics are: paralysis is more limited. Due to the interruption of the reflex arc, the flaccid muscles of the paralyzed limbs were flaccid paralysis. The damage of the efferent pathway of reflex arc leads to the decrease or disappearance of tendon reflex. No pathological reflex occurred. Because of the conduction of motor nerve obstruction to some part of the muscle fiber waste, and the release of acetylch