impact on the expression of the nectar of disinfection dan of the rat tlr4 damp-heat syndrome febrile disease of nf κbp65 in（影响表达的甘露消毒丹鼠tlr4湿热综合症的nfκbp65发热性疾病）.docVIP

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2017-09-06 发布于浙江
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impact on the expression of the nectar of disinfection dan of the rat tlr4 damp-heat syndrome febrile disease of nf κbp65 in（影响表达的甘露消毒丹鼠tlr4湿热综合症的nfκbp65发热性疾病）.doc

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impact on the expression of the nectar of disinfection dan of the rat tlr4 damp-heat syndrome febrile disease of nf κbp65 in（影响表达的甘露消毒丹鼠tlr4湿热综合症的nfκbp65发热性疾病）

Impact on the expression of the nectar of disinfection Dan of the rat TLR4 damp-heat syndrome febrile disease of NF κBp65 in [Abstract] observed nectar disinfection Dan on damp-heat syndrome rat cells febrile disease endotoxin Toll-like receptor 4 (TCR4) mRNA and nuclear factor kappa B (NF κB) p65 expression changes of therapeutic mechanism of heat dampness party. Methods] Wistar rats were randomly divided into normal group, hot and humid model group (high fat + high temperature and humidity + E. coli heat dampness group, by reverse transcription - polymerase chain reaction (RT PCR detection of liver tissue macrophages TLR 4mRNA, immunohistochemical detection of liver tissue macrophage NF κBp65 in activation. [results] model group 6,12,24 h of infection phase point of TLR4 of NF  κBp65 expression gradually increased, and no comparison between time points are There are significant differences (P lt;0.05 or P lt;0.01, with the course of development, hot and humid model TLR4, NF κBp65 in expression increased the treatment group 6,12,24 h time points TLR4mRNA expression decreased gradually: 12,24 h time point treatment group and the model group of TLR4 expression significantly with resistance weakened (P lt;0.05, P lt;0.01, but with the normal group comparison there is still significant with difference (P lt;0.01 Description TLR4mRNA curb decay is a slow the process. while NF κBp65 6,12,24 h activation at each time point of the treatment group showed no significant difference compared with the normal group, no significant changes in the model group, but at the same time point, there were significant differences (P lt;0.05 or P lt;0.01. [Conclusion] mannose disinfection Dan of febrile disease damp-heat syndrome rat liver macrophages TLR4mRNA, and NF κBp65, expression the better intervention role, to suspend the transcription of inflammatory mediators, limit the acute inflammatory response. [Keywords] the the Ganlu Qing poison immortality Science damp-heat syndro