IBHRE复习资料：药物和起搏器试题.docxVIP

1. Beta-blocker (class II) therapy acts by which of the following? a. Decrease sympathetic tone affecting mainly the SA and AV node b. Increases sympathetic tone which increases cardiac contractility c. Blockage of calcium channels d. Has an unknown mechanism of action but increases cardiac contractility Ans : ________ Antiarrythmic agents (class I) are broken down into three categories. Which class slows the upstroke of the action potential, prolongs the duration, and increase refractoriness? a. Class Ia b. Class Ib c. Class Ic d. Class IV Ans : ______ 3. What class of drug is Digitalis? Class I Class II Class IV Class V Ans : ________ 4. What effect does Diltiazem have on the AV node? Slows conduction by blocking calcium channels Prolongs the refractory period by blocking sodium channels Increases the HV interval by blocking the calcium channels None of these Ans : ______ 5. Which of the following drugs would not interact with Amiodarone? Coumadin (warfarin) Digoxin Quinidine Sotalol Ans : ________ After being started on antiarrhytmic therapy, your patient develops nausea, vomiting, and diarrhea. Which of the following drugs is most commonly associated with these side effects? Digoxin Verapamil Procainamide Quinidine Ans : ______ Which type of medication has been proven to decrease the incidence of sudden cardiac death in the post MI patient? a. ACE inhibitor b. Beta blocker c. Calcium channel blocker d. Sodium channel blocker Ans : ______ Knowing the Vaughn-Williams class anti-arrhythmics, which of the following is not a CLASS I? Disopyramide Lidocaine Quinidine Sotalol Ans : ________ What medication is considered to be first line treatment of torsade de pointes? Atropine Lidocaine Epinephrine Isuprel Ans : ______ A pacemaker dependent patient presents to the hospital with near-syncope. Telemetry confirms loss of capture. The pat