Extravascular lung water and intrathoracic blood volume determination in guiding significance for diagnosis and treatment of ARDS patients.docVIP

 PAGE \* MERGEFORMAT 21 Extravascular lung water and intrathoracic blood volume determination in guiding significance for diagnosis and treatment of ARDS patients Keywords: extravascular lung water in patients with ARDS Acute respiratory distress syndrome (acute respiratory distress syndrome, ARDS) occurred in the severe infections, shock, trauma and burns and other disease processes, due to pulmonary capillary endothelial cells and alveolar epithelial cell injury caused by diffuse interstitial and alveolar pulmonary edema, and caused to progressive hypoxemia, respiratory distress as a clinical syndrome characterized by [1]. High-permeability pulmonary edema was the pathophysiological features of ARDS, pulmonary edema, the extent and prognosis of ARDS was positively correlated. Thus, through active fluid management, improved ARDS patients with pulmonary edema is an important clinical significance. Recently, different fluid management strategies ARDS studies have shown that, despite the restrictive fluid management and non-restrictive fluid management group no significant difference in mortality, but non-restrictive fluid management compared to restrictive fluid management (diuretic and limitations rehydration) Section the first week of the fluid balance in patients with a negative balance of [- (136 ± 491) ml compared with (6992 ± 502) ml], oxygenation index significantly improved lung injury score was significantly reduced, and ICU length of stay was significantly shorter. Of particular note is that restrictive fluid management group of the incidence of shock and hypotension is not significantly increased. Can be seen in the maintenance cycle stability, and ensure that under the premise of organ perfusion, restrictive fluid management strategy is beneficial for ARDS patients [2]. How to provide sufficient capacity for patients in order to ensure recovery, while avoiding or aggravating hemodynamic pulmonary edema is the managemen