Urinary TWEAK as a biomarker of lupus nephritis a multicenter cohort study 英文参考文献.docVIP

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Urinary TWEAK as a biomarker of lupus nephritis a multicenter cohort study 英文参考文献.doc

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Urinary TWEAK as a biomarker of lupus nephritis a multicenter cohort study 英文参考文献

Available online /content/11/5/R143 Research article Open Access Vol 11 No 5 Urinary TWEAK as a biomarker of lupus nephritis: a multicenter cohort study Noa Schwartz1,2, Tamar Rubinstein1, Linda C Burkly3, Christopher E Collins4,5, Irene Blanco1, Lihe Su3, Bernard Hojaili1, Meggan Mackay6, Cynthia Aranow6, William Stohl4, Brad H Rovin7, Jennifer S Michaelson3 and Chaim Putterman1,8 1Division of Rheumatology, Albert Einstein College of Medicine, 1300 Morris Park Avenue, Bronx, NY 10461, USA 2Hadassah University Hospital, POB 12000, Ein Kerem, Jerusalem 91120, Israel 3Biogen Idec, 14 Cambridge Center, Cambridge, MA 02142, USA 4Department of Medicine, Division of Rheumatology, Los Angeles County + University of Southern California Medical Center and University of Southern California, Keck School of Medicine, 2011 Zonal Avenue, Los Angeles, CA 90033, USA 5Washington Hospital Center, 110 Irving Street, NW, Washington, DC 20010, USA 6Feinstein Institute for Medical Research, 350 Community Drive, Manhasset, NY 11030, USA 7Ohio State University Medical Center, 410 West 10th Avenue, Columbus, OH 43210, USA 8Department of Microbiology and Immunology, Albert Einstein College of Medicine, 1300 Morris Park Avenue, Bronx, NY 10461, USA Corresponding author: Chaim Putterman, putterma@ Received: 4 Aug 2009 Revisions requested: 9 Sep 2009 Revisions received: 21 Sep 2009 Accepted: 28 Sep 2009 Published: 28 Sep 2009 Arthritis Research Therapy 2009, 11:R143 (doi:10.1186/ar2816) This article is online at: /content/11/5/R143 ? 2009 Schwartz et al.; licensee BioMed Central Ltd. This is an open access article distributed under the terms of the Creative Commons Attribution License (/licenses/by/2.0), which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. Abstract Introduction TNF-like weak inducer of apoptosis (TWEAK) has been implicated as a mediator of chronic inflammatory processes via prolong