头 痛(国外英语资料).doc

头 痛 The thirteenth chapter is headache (Heaclache) an overview of the first section Headache (headache) is one of the most common symptoms of the disease, and its pathogenesis is very complicated. The intracranial lesions; such as brain tumors, cerebral hemorrhage, subarachnoid hemorrhage, cerebral edema, meningitis, brain abscess, intracranial hypertension, cerebral arteritis and phlebitis can cause; the functional or mental illness may cause, such as: extracranial vessel dilatation caused by migraine, tension in the neck muscles the headache and cranial contraction, and common clinical neurotic headache; systemic diseases such as fever after the hypoxia, hypoglycemia, carbon monoxide poisoning and dysentery by big attack, sinusitis, amblyopia and ametropia, the use of vasodilators also can cause headaches. Including 1. head pain sensitive structure: the scalp, subcutaneous tissue, galea and periosteum; the head and neck vessels and muscle; the skull base artery and its branches, dural artery, intracranial venous sinus and its main branches; the trigeminal and glossopharyngeal, vagus nerve and ganglion nerve and neck 2 - 3. The upper part of the tentorium innervated by the trigeminal nerve, the lesion is mainly caused by the face, forehead, and scoop top front pain; lower tentorium (posterior fossa) dominated by glossopharyngeal, vagus nerve and neck 2 - 3 nerve lesions in this area is mainly caused by the occiput, ear and ear pharyngeal pain. The brain tissue itself without sensory nerves, is most of the dura mater, ependyma, meninges, choroid plexus, pial vein, small intracranial vessels and skull with little or no sensory nerve fibers, not sensitive to pain. If the pain sensitive structure of the head is stimulated, oppression, or higher nervous activity car disorders can cause pain; head and neck muscle sustained contraction, arterial dilatation, contraction or displacement of cranial nerve and cervical nerve compression, injury or chemical stimulation are the