carbonic anhydrases iii and iv autoantibodies in rheumatoid arthritis, systemic lupus erythematosus, diabetes, hypertensive renal disease, and heart failure碳酸脱水酶iii和iv自身抗体在类风湿性关节炎、系统性红斑狼疮、糖尿病、高血压肾脏疾病,心力衰竭.pdfVIP

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carbonic anhydrases iii and iv autoantibodies in rheumatoid arthritis, systemic lupus erythematosus, diabetes, hypertensive renal disease, and heart failure碳酸脱水酶iii和iv自身抗体在类风湿性关节炎、系统性红斑狼疮、糖尿病、高血压肾脏疾病,心力衰竭.pdf

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carbonic anhydrases iii and iv autoantibodies in rheumatoid arthritis, systemic lupus erythematosus, diabetes, hypertensive renal disease, and heart failure碳酸脱水酶iii和iv自身抗体在类风湿性关节炎、系统性红斑狼疮、糖尿病、高血压肾脏疾病,心力衰竭

Hindawi Publishing Corporation Clinical and Developmental Immunology Volume 2012, Article ID 354594, 6 pages doi:10.1155/2012/354594 Clinical Study Carbonic Anhydrases III and IV Autoantibodies in Rheumatoid Arthritis, Systemic Lupus Erythematosus, Diabetes, Hypertensive Renal Disease, and Heart Failure Chengeng Liu,1 Yue Wei,2 Jianmin Wang,3 Langan Pi,3 Jianjun Huang,3 and Peichang Wang1 1 Department of Clinical Laboratory, Xuanwu Hospital, Capital Medical University, 45 Changchun Road, Beijing 100053, China 2 Department of Oncology, Dongfang Hospital, Beijing University of Chinese Medicine, Beijing 100078, China 3 Department of Clinical Immune, Xiangya School of Medicine, Central South University, Changsha 410078, Hunan, China Correspondence should be addressed to Peichang Wang, peichangwang@ Received 8 July 2012; Accepted 14 August 2012 Academic Editor: Dimitrios Bogdanos Copyright © 2012 Chengeng Liu et al. This is an open access article distributed under the Creative Commons Attribution License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly cited. In the present study, the CA III and IV autoantibodies, CA activity, antioxidant enzymes and cytokines in rheumatoid arthritis (RA), systemic lupus erythematosus (SLE), diabetes, hypertensive renal disease, and heart failure were investigated. The anti-CA III antibody titers in patients with RA, SLE, and type 1 diabetes (T1D) were signiﬁcantly higher than that in control groups (P 0.05). The anti-CA IV antibody titers in patients with RA, SLE, type 1 diabetic nephropathy (T1DN), and heart failure were signiﬁcantly higher than that in control groups (P 0.05) while anti-CA IV antibody could suppress the total CA activity. The SOD and GPx