acute carbon monoxide poisoning in pre-hospital emergency care（在院前急救护理急性一氧化碳中毒）.docVIP

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acute carbon monoxide poisoning in pre-hospital emergency care（在院前急救护理急性一氧化碳中毒）.doc

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acute carbon monoxide poisoning in pre-hospital emergency care（在院前急救护理急性一氧化碳中毒）

Acute carbon monoxide poisoning in pre-hospital emergency care 1 Materials and Methods 1.1 General Information January 2008 to December 2010, I treated 72 cases of acute carbon monoxide poisoning, 25 cases were male and female 47 cases; age from 4 to 76 years, an average of 32.4 years. Coal stove for heating in 46 cases (63.9%, gas water heater shower poisoning in 10 cases (13.9%, gas stove improper use of poisoning in five cases (6.9%, suicide in four cases (5.6%, the scene of death in 7 cases (9.7% according to the degree of intoxication classification Standard: 17 cases of mild to moderate in 28 cases, 27 cases, severe. 1.2-site first aid and care Immediately from the first aid environment, as soon as possible so that the poisoning scene with the outside ambient air circulation, poisoning patients quickly go to the fresh air and well-ventilated place, unlock the collar, to keep warm, supine, head to side side to maintain airway patency, timely and clear vomit and secretions in the respiratory tract, to prevent the occasional lead to suffocation of the heart, respiratory arrest patients, immediate cardiopulmonary resuscitation. quickly to correct hypoxia, coma or irritability immediately unlock the collar, to clear the respiratory secretions in a timely manner to ensure that the airway Meanwhile, according to the condition of mild poisoning were given high-flow nasal cannula oxygen, severe poisoning, give oxygen mask, critically ill patients with tracheal intubation easy breathing bag or ventilator-assisted breathing. the rapid establishment of intravenous access, rescue medication in order to facilitate the intravenous injection, to gain time for successful treatment. effective drug treatment is the prevention and treatment of brain edema and improved cerebral tissue hypoxia, and effective measures to restore the function of brain cells. 2 to 4 hours after acute poisoning, cerebral edema, 24 to 48 is the development of brain edema peak hours, as