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- 约4.38万字
- 约 40页
- 2019-01-30 发布于广东
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Abstract
andmodel3d
group group(P0.05).
9、NSE ld
integratedopticaldensity(IOD)increasedgradually.Modelgroup
was lessthan
thatatmodel3d and21d
group,7d
significantly group,14dgroup
21d was thanthoseatmodel3d and
group.Modelgroupsignificantlygreater group
model7dgroup(P0.05).
Conelusion:
After firstinflammationandthenleadsto
MCAO,before
dayl4,at aggravated
and cell ofneuralstemcell and
apoptosisglial proliferation,therepair proliferation
differentiationstarted.After cerebralinfarctionareaandthebrain
dayl4although
wasstill to and
damage increasing,resistanceapoptosis enhanced
significantly
factorsincreasedresultin ofnervecells
neurotrophic partialischemia/hypoxia
half
dark and ofcellular stem
(ischemiaarea)be
protectedrecovery function,neural
cells anddifferentiation
causenerveeell nervefunction
proliferation regeneration.So
after 4.
improvedsignificantly
dayl
VI
目录
目录
第1章前言………………………………………………………………………….1
第2章材料与方法…………………………………………………………………3
2.1实验动物及分组……………………
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