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NO ET-1 in patients with obstructive sleep apnea-hypopnea syndrome in Progress
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NO ET-1 in patients with obstructive sleep apnea-hypopnea syndrome in Progress
[Keywords:] Obstructive sleep apnea-hypopnea syndrome NO ET-1
Obstructive sleep apnea-hypopnea syndrome (Obstructive sleep apnea-hypopnea syndrome, OSAHS) is a common sleep breathing disorders, it is because during sleep in patients with recurrent partial or complete upper airway obstruction caused by apnea and hypoxia hyperlipidemia, sleep structure, disorder, common in the elderly, but now there are getting younger and younger, young adults view physical health appears severely damaged. Clinically, in order to habitual snoring, daytime sleepiness and merging different degrees of organ dysfunction as the main performance [1]. Modern medicine in many disorders that OSAHS has become the source of the disease, and the impact has been seen as major cause of death. OSAHS patients during sleep apnea caused by repeated episodes of hypoxemia, awakening to reflect and sharp fluctuations in intrathoracic negative pressure through the neuro-hormonal factors such as long-term effects caused by endothelial dysfunction. NO, ET-1 is a release of vascular endothelial small molecules, both the maintenance of vasomotor function of the interaction. Study confirmed that, OSAHS is essential hypertension (EH) is an independent risk factor for disease [2]. Repeated sleep apnea can cause high blood pressure, high blood pressure and hypoxia-related. Clinical treatment is aimed at alleviating symptoms of snoring and apnea, hypoxemia lift will help to control and gradually restore the body in patients with metabolic functions.
1 OSAHS treatment status
Obstructive sleep apnea-hypopnea syndrome (OSAHS) is the most common clinical disease of sleep-disordered breathing (SDB) is one, OSAHS with snoring, repeated apnea (hypopnea) and the consequent awakening (arousal), lead to inadequate sleep at night, and varying degrees of daytime sleepiness such as the main symptom, and can lead to
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