Qi Fever Syndrome in Rabbits skin lung spleen morphology.doc

Qi Fever Syndrome in Rabbits skin lung spleen morphology.doc

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Qi Fever Syndrome in Rabbits skin lung spleen morphology

 PAGE \* MERGEFORMAT 13 Qi Fever Syndrome in Rabbits skin lung spleen morphology Of: Lin Man, Wang Hui, Liu Wenbin, Tang Xiaofeng [Abstract] Objective Qi Fever Syndrome in Rabbits of the skin, lungs, spleen, morphological changes of Qi heat to the skin, lungs and spleen. Methods Rabbits were divided into 2 groups, control group and Qi fever group Fasting can not help with water and intraperitoneal injection of lipopolysaccharide (LPS) Fever build Qi, take the skin, spleen, lung tissues were paraffin tissue sections, the morphological changes observed under the microscope. The results observed under the microscope tissue sections, compared with the normal group , Qi fever in rabbits with inflammatory cell infiltration of local lung, spleen and local organizations like multinucleated giant cell tumor infiltration and see granulomatous disease, irregular skin wrinkled skin, hair follicles shorter, significantly reduced the number of pores first. Conclusion Qi Fever Syndrome in Rabbits of the skin, spleen, lungs compared with normal rabbits there morphological differences. [Keywords:] Qi fever; skin; lung; spleen; morphology Abstract: ObjectiveTo investigate the morphologic changes in skin, lung and spleen of rabbits with syndrome of fever due to qi deficiency, and to explore the effect of syndrome of fever due to qi deficiency on skin, lung and spleen. Methods Rabbits were randomly divided into two groups, normal group and group of syndrome of fever due to qi deficiency. The syndrome of fever due to qi deficiency group was developed by administering LPS and fasting. Paraffin sections of skin, lung, and spleen were prepared after hematoxylin and eosin (HE ) staining, and morphological changes were observed under light microscopy. Results Compared with the normal tissue sections from the control group, local inflammatory cell infiltration from lung tissue, infiltration of focal multinucleated giant cells and granulomatous lesion from spleen, and irregula

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