原创力文档cardiac-autonomic imbalance and baroreflex dysfunction in the renovascular angiotensin-dependent hypertensive mouse心脏自主失衡在肾血管性高血压老鼠angiotensin-dependent和压力反射功能障碍.pdfVIP
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cardiac-autonomic imbalance and baroreflex dysfunction in the renovascular angiotensin-dependent hypertensive mouse心脏自主失衡在肾血管性高血压老鼠angiotensin-dependent和压力反射功能障碍
Hindawi Publishing Corporation
International Journal of Hypertension
Volume 2012, Article ID 968123, 9 pages
doi:10.1155/2012/968123
Review Article
Cardiac-Autonomic Imbalance and Baroreflex Dysfunction in
the Renovascular Angiotensin-Dependent Hypertensive Mouse
Bianca P. Campagnaro,1 Agata L. Gava,2 Silvana S. Meyrelles,1, 2 and Elisardo C. Vasquez1, 3, 4
1 Department of Physiological Sciences, Health Sciences Center, Federal University of Espirito Santo, 29075-910 Vitoria, ES, Brazil
2 Biotechnology Graduate Program, Health Sciences Center, Federal University of Espirito Santo, 29075-910 Vitoria, ES, Brazil
3 Department of Physiological Sciences, Emescam College of Health Sciences, 29045-402 Vitoria, ES, Brazil
4 Lab Transgenes and Cardiovascular Control, Department of Physiological Sciences, Health Sciences Center,
Federal University of Espirito Santo, Avenida Marechal Campos 1468, 29042-755 Vitoria, ES, Brazil
Correspondence should be addressed to Elisardo C. Vasquez, evasquez@.br
Received 3 September 2012; Accepted 10 October 2012
Academic Editor: Ovidiu C. Baltatu
Copyright © 2012 Bianca P. Campagnaro et al. This is an open access article distributed under the Creative Commons Attribution
License, which permits unrestricted use, distribution, and reproduction in any medium, provided the original work is properly
cited.
Mouse models provide powerful tools for studying the mechanisms underlying the dysfunction of the autonomic reflex control
of cardiovascular function and those involved in cardiovascular diseases. The established murine model of two-kidney, one-
clip (2K1C) angiotensin II-dependent hypertension represents a useful tool for studying the neural control of cardiovascular
function. In this paper, we discuss the main contributions from our
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